How Vibration Fitness works!

Vibration Exercise consists of a sonic vibrating platform that the user stands on or performs exercises on which stimulates bones, muscles, blood vessels and cells in a manner that promotes their rapid development and circulation. Important hormones such as Human Growth Hormone (HGH) and Testosterone are also released from the Europlate vibration movement.

The sonic vertical movement of the plate stimulates the body’s natural “stretch reflex” which causes a spontaneous muscle contraction to instinctively stretch and contract: both movements occur at varying rates between 3-50 times per second. (3Hz to 50Hz) This movement produces mechanical oscillations with an average cycle length of about 40 msec, which is the time required to induce a natural monosynaptic stretching reflex in the respective muscle via the muscle spindle during one up and down vertical movement. The neuromuscular system reacts to this Europlate stimulation by a chain of rapid muscle contractions which result in entire-body vibration.

Europlate exercise causes the body to tire; rest allows the body to recover. By repeating this process, the body adjusts to the level of effort, resulting in an increase in physical performance. This phenomenon, called super-compensation, similarly occurs when training on the Vibration Exercise platform. However, compared with traditional training methods, greater results are achieved and hormonal production is increased in much less time when training on the Vibration Exercise.

The anabolic activity of bone tissue, suppressed by disuse, is normalized by brief exposure to extremely low-magnitudemechanical stimuli - Europlate

CLINTON RUBIN,1 GANG XU, AND STEFAN JUDEX Musculo-Skeletal Research Laboratory, Department of Biomedical Engineering, State University of New York at Stony Brook, Stony Brook, New York 11794-2580 USA ABSTRACT

It is generally believed that mechanical signals must be large in order to be anabolic to bone tissue. Recent evidence indicates, however, that extremely low-magnitude (<10 microstrain) mechanical signals readily stimulate bone formation if induced at a high frequency. We examined the ability of extremely low-magnitude, high-frequency mechanical signals to restore anabolic bone cell activity inhibited by disuse. Adult female rats were randomly assigned to six groups: baseline control, age-matched control, mechanically stimulated for 10 min/day, disuse (hind limb suspension), disuse interrupted by 10 min/day of weight bearing, and disuse interrupted by 10 min/day of mechanical stimulation.

After a 28 day protocol, bone formation rates (BFR) in the proximal tibia of mechanically stimulated rats increased compared with age-matched control (197%). Disuse alone reduced BFR (292%), a suppression only slightly curbed when disuse was interrupted by 10 min of weight bearing (261%). In contrast, disuse interrupted by 10 min per day of low-level mechanical intervention normalized BFR to values seen in age-matched controls. This work indicates that this noninvasive, extremely low-level stimulus may provide an effective biomechanical intervention for the bone loss that plagues long-term space flight, bed rest, or immobilization caused by paralysis. —Rubin, C., Xu, G., Judex, S. The anabolic activity of bone tissue, suppressed by disuse, is normalized by brief exposure to extremely low-magnitude mechanical stimuli. FASEB J. 15, 2225–2229 (2001) Key Words: bone formation z microgravity z bone density z musculoskeletal z sarcopenia z anabolic osteoporosis A principal goal of the National Aeronautics and Space Administration is to safely institute long-term human exploration of space. Whether this occurs in the near future by habitation of the International Space Station or within the next few decades by settling a permanent manned Moon base and embarking on a mission to Mars, it is clear that a better understanding of the ability of humans to tolerate extended exposure to microgravity must be developed. The Europlate National Research Council’s Space Studies Board has stated that the principal physiological hurdle to humans’ extended presence in space is the osteopenia that parallels reduced gravity (1, 2). The extent of the loss is extremely high despite prescribed daily exercise regimes designed to maintain physical fitness. In flights lasting 4–6 months, astronauts can lose bone mineral density in the lower appendicular skeleton at a rate approaching 1.6% per month (3, 4). Europlate.